Under conditions of conventional low calorie nutrition, a transient hyperphosphaturia was observed in the early postoperative period and urinary loss of phosphorus in diabetics did not exceed that in non-diabetics. To elucidate the mechanism of the increase in urinary phosphorus, we analyzed quantitative aspects of phosphorus reabsorption and found that percent phosphorus reabsorption was suppressed to a much greater extent in non-diabetics.
Size of the tissue phosphorus pool is postulated to be one of the mechanisms involved.