HIF-1-mediated suppression of mitochondria electron transport chain function confers resistance to lidocaine-induced cell death

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Use this link to cite this item : https://ir.lib.shimane-u.ac.jp/53342

language	eng
Author	Okamoto Akihisa Department of Anesthesiology, Kansai Medical University, Hirakata, Japan Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan View author publications Sumi Chisato Department of Anesthesiology, Kansai Medical University, Hirakata, Japan Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan Tanaka Hiromasa Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan Kusunoki Munenori Department of Anesthesiology, Kansai Medical University, Hirakata, Japan Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan Iwai Teppei Department of Anesthesiology, Kansai Medical University, Hirakata, Japan Nishi Kenichiro Department of Anesthesiology, Kansai Medical University, Hirakata, Japan Matsuo Yoshiyuki Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan Harada Hiroshi Laboratory of Cancer Cell Biology, Radiation Biology Center, Kyoto University, Kyoto, Japan Precursory Research for Embryonic Science and Technology (PRESTO), Japan Science and Technology Agency (JST), Saitama, Japan Takenaga, Keizo Bono Hidemasa Database Center for Life Science (DBCLS), Research Organization of Information and Systems (ROIS), Mishima, Japan Hirota Kiichi Department of Human Stress Response Science, Institute of Biomedical Science, Kansai Medical University, Hirakata, Japan
Journal Title	SCIENTIFIC REPORTS
Volume	7
ISSN	2045-2322
Published Date	2017-06-19
DOI	10.1038/s41598-017-03980-7
PubMed ID	28630416
Publisher	NATURE PUBLISHING GROUP
NII Type	Journal Article
OAI-PMH Set	Faculty of Medicine
Remark	This work was supported by the Japan Society for the Promotion of Science KAKENHI; Grants #26670693 and #24592336 to K.H., #25462457 to K.N., and #15K15577 to T.I. This work was also supported by the National Bioscience Database Center (NBDC) of the Japan Science and Technology Agency (JST) to H.B. We would like to thank Editage (www.editage.jp) for English language editing.