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language
eng
Author
Rahman Mohammed Tanjimur Departments of Obstetrics and Gynecology, Shimane University School of Medicine, Enyacho 89-1, Izumo, Shimane 6938501, Japan
Rahman Munmun Departments of Obstetrics and Gynecology, Shimane University School of Medicine, Enyacho 89-1, Izumo, Shimane 6938501, Japan
Katagiri Hiroshi Departments of Obstetrics and Gynecology, Shimane University School of Medicine, Enyacho 89-1, Izumo, Shimane 6938501, Japan
Katagiri Atsuko Departments of Obstetrics and Gynecology, Shimane University School of Medicine, Enyacho 89-1, Izumo, Shimane 6938501, Japan
Nakayama Naomi Department of Biocehmistory, Shimane University School of Medicine, Izumo, Shimane 6938501, Japan
Ishikawa Noriyuki Department of Organ Pathology, Shimane University School of Medicine, Izumo, Shimane 6938501, Japan
Miyazaki Kohji Departments of Obstetrics and Gynecology, Shimane University School of Medicine, Enyacho 89-1, Izumo, Shimane 6938501, Japan
Subject
type II ovarian carcinoma; KRAS; MAPK1; gene amplification; survival; MEK inhibitor
Journal Title
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume
14
Issue
7
Start Page
13748
End Page
13762
ISSN
1422-0067
Published Date
2013-07
DOI
PubMed ID
Publisher
MDPI AG
NII Type
Journal Article
OAI-PMH Set
Faculty of Medicine
Remark
This study was supported by grants from the Ministry of Education, Culture, Sports, Science, and Technology in Japan.